Gout is an ancillary condition of your body protecting against glucose poisoning. Glucose poisoning occurs from habitual consumption of too much sugar and processed food, a proxy of sugar. Gout usually diminishes as your glucose condition worsens, thus when gout goes away, it can be a signal of diabetes.
When your benelles become glucose saturated, they refuse more glucose, in order to protect their health. To tell the blood “no more sugar please”, they refuse insulin. The term for this is insulin intolerance.
But the sugar has to go somewhere; elevated blood sugar is harmful, and benelles actively resist harm. Liver and fat cells will absorb extra glucose, helping reduce blood sugar. But if more glucose keeps coming, the liver and fat cells will also become insulin intolerant and stop absorbing glucose.
At this point, sugar and insulin are knocking on every door, and no one is letting them in. This “no vacancy” leads to dangerously high blood sugar levels. The excess circulating sugar starts to combine with proteins that it shouldn’t, like LDL cholesterol.
“Sugarized” LDL forms a sludge or “gunk” that clogs, scars, narrows and hardens blood vessels. Benelles respond with inflammatory macrophage attack, to clear the sludge and preserve the health of veins and arteries. But still, the patient sticks to improper diet, and floods her system with more sugar.
Suddenly, there’s a big problem. The blood is full of sugar, causing damage; plus, no one’s taking sugar and macrophages are fighting a losing battle in the blood vessels. By this time, the pancreas understands no more glucose is needed and reduces insulin. This is not, as current medical professionals think, “insufficient insulin production”; it is correct insulin production, given the circumstances.
At this point, the surplus glucose is essentially quarantined in the blood, with the kidneys allowing excess glucose to pass into urine. Yes, the glucose is toxic in the blood, but it would be worse to force-feed it into tissue; the pancreas knows this. Plus, there is a special kidney mechanism for handling blood sugar poisoning. The kidneys, while working to excrete excess sugar—a relatively slow, labor intensive process—also employ a more immediate power to counteract glucose damage to blood vessels: uric acid.
Kidneys increase uric acid levels in the blood stream to reduce damage from the glucose. Uric acid is a potent antioxidant, estimated to contain half the antioxidant capacity of blood plasma. For those on processed food diets, devoid of antioxidants, uric acid is acting like the antioxidants that the body expected in food. Essentially, uric acid is medicine for preventing diabetes. Here, diabetes should be thought of as a circulatory disease from advanced blood vessel damage from chronic sugar poisoning.
Gout occasionally occurs when kidneys raise uric acid levels to a high level in order to prevent blood vessel damage and diabetes. Yes, gout is painful, but it’s not a serious condition when compared with blood vessel damage or diabetes. Gout is incidental and necessary to protect long-term health. Uric acid is not the enemy, but the friend. Unfortunately, uric acid can crystallize in areas of less circulation and lower temperature, like big toes.
An elevated uric acid level prevents diabetes by protecting your heart, blood vessels, and kidneys against damage from sugar poisoning. In a sense, gout is a sign of health. But at this point, conventional medicine tries to reduce uric acid levels to stop gout. Certainly, lower uric acid might decrease gout, but at the cost of advancing diabetes; a poor long-term decision.
Uric acid and macrophage clean-up help prevent diabetes, however if the patient continues with sugar inundation, there will be accumulation of blood vessel damage, eclipsing into diabetes.
Once diabetes sets in, gout is unlikely because uric acid levels drop off. At the same time, as diabetes comes, the protein SGLT2 will upregulate, causing the kidneys to release more glucose into glucose-poisoned blood.
This kidney behavior is not, as current medicine describes, “defective renal glucose handling”. Rather, the blood vessel damage has become so extensive by this point that some tissues around the body are literally starving from lack of blood flow and glucose, in many cases simultaneous with a pot belly. It is starving cells next to fatty cells. This is the essential calamity of diabetes; the kidneys are forced to make too-sweet blood even sweeter, so that impaired tissue—cutoff by blood vessel death—can take tiny, concentrated sips of glucose in order to survive. An analogy is a pulmonary patient taking little breaths on an oxygen tank.
Although a kidney-induced, super-sweetening of the blood increases the severity of diabetes, it also keeps immediately-dying tissue alive, which is the correct long-term calculation by benelles. Here, the kidneys are sacrificing their life to keep other benelles alive. The extra sweet blood will systematically destroy the kidneys until they fail.
Uric acid is not the cause of hyperuricemia or diabetes; it protects against them. Gout is useful in that it reveals three things: your kidneys are still essentially healthy, you have sugar poisoning, and you do not yet have advanced blood vessel damage. The proper diagnosis of diabetes should be “when uric acid levels taper”, as this indicates blood vessel damage sufficiently severe that the kidneys recalculate toward suicide to save remaining tissue around the body. The pathways for how the kidneys understand when to make this calculation should be studied.
In short, gout is a sign of healthy kidneys which are protecting sick blood vessels. Patients should be advised not to lower uric acid with drugs, as this will allow more rapid deterioration of blood vessels, leading to faster onset of diabetes.
Gout is predictive of diabetes only in the sense that it tells us one’s eating habits are causing diabetes. In fact, gout is a footprint left by a guardian angel, fighting the demon of diabetes. Processed food habits must be changed, or there won’t be any footprints.
Natural food and drink will reduce insulin intolerance and bring down blood sugar levels, reversing gout and diabetes. Of special consideration are cinnamon and coffee. Six cups of coffee a day substantially reduces uric acid level. This is because it increases cellular metabolism across the body, allowing extra glucose burn—even without exercise. Effectively, this shifts glucose from the blood into cells, thereby preventing the kidneys from needing to elevate the uric acid level to protect the blood from oxidative stress.
Natural foods break into sugar at a natural rate, so are unlikely to cause chronic sugar poisoning. The antioxidants in natural foods allow them to circulate in your blood vessels for long periods without causing damage, so even if you over-eat, there is a built-in protective mechanism in the food itself. On the other hand, processed fructose starts causing damage right away. Thus, today’s problem isn’t as much food abundance; it’s that our food and drink are made of cheap starch and artificial chemicals that break down in rapid and toxic ways, peculiar to pancreas and kidney benelles. Never before have men and women been able to sustain a constantly high sugar level, making gout a disease of affluence in the context of human history. But today, even the homeless drink soda; it’s eating natural foods with their protective antioxidants that requires affluence, meaning gout is, in our times, a disease of poverty and poor eating habits.
On a bright note, even the poor might follow the outstanding medical advice once given to the world’s richest man, John Rockefeller. The advice is so effective it might be used to stay healthy, even on a processed food diet: avoid worry, get plenty of mild exercise, always stop eating while you’re still a little hungry.
What’s next? Probably, kidney benelles will evolve to see sugar as more of a plentiful poison instead of a precious resource. They may develop a special ability to allow rapid sugar urination, thus controlling sugar poisoning. Or maybe our blood vessels will develop some special slippery, or regenerative, property which makes high blood sugar less relevant, allowing for turbocharged periods of sugar-induced, mental performance. Either mutation would be a perfect next step for a world that lives on cheap chemicals and sugars.
Discusses how free radicals are formed disproportionately in diabetes by glucose oxidation:
Maritim AC, et al “Diabetes, oxidative stress, and antioxidants: a review.” J Biochem Mol Toxicol. 2003;17(1):24-38 doi: 10.1002/jbt.10058
Discusses the strong antioxidant effect of uric acid:
Sautin Y, Johnson R “URIC ACID: THE OXIDANT–ANTIOXIDANT PARADOX” Nucleosides Nucleotides Nucleic Acids. 2008 Jun; 27(6): 608–619. doi: 10.1080/15257770802138558
Becker B “Towards the physiological function of uric acid”. Free Radical Biology & Medicine 14 (6): 615–31. doi: 10.1016/0891-5849(93)90143-I
Shows pre-diabetics have increased uric acid levels:
Dehghan A, et al “High Serum Uric Acid as a Novel Risk Factor for Type 2 Diabetes” Diabetes Care 31:361–362, 2008 weblink
Mistakenly asserts that uric acid predicts diabetes, when it is actually poor eating habits like consuming fructose that predict diabetes—while uric acid prevents:
Katsiki N, et al “Uric acid and diabetes: Is there a link?” Curr Pharm Des. 2013;19(27):4930-7. weblink
Johnson R, et al “Sugar, Uric Acid, and the Etiology of Diabetes and Obesity” Diabetes. 2013 Oct; 62(10): 3307–3315. doi: 10.2337/db12-1814
Shows diabetics have reduced uric acid levels:
Bandaru P, Shankar A “Association between Serum Uric Acid Levels and Diabetes Mellitus” Int J Endocrinol. 2011;2011:604715. doi: 10.1155/2011/604715
Shows 6 cups of coffee a day reduces uric acid level:
Choi H, Curhan G “Coffee, tea, and caffeine consumption and serum uric acid level: the third national health and nutrition examination survey.” Arthritis Rheum. 2007 Jun 15;57(5):816-21. doi 10.1002/art.22762
This article is an example of how the Benelles philosophy trusts good cells to make the best long-term decisions. Frequently, when we face a painful condition, we want a doctor to relieve the immediate symptom. Doctors often do, at the expense of long-term health. For instance, with gout, doctors try to bring down uric acid levels, putting you at greater risk of diabetes and kidney failure. The end result is lots of pain-free big toes—and dialysis clinics all over town.
Benelles sees health events like gout as signs of what is going on, and how the body is coping with adverse conditions. The goal is to understand the artificial paths of stress and damage we have put upon our benelles, then change the conditions to be favorable to benelles.
Government endorsement of conventional medicine effectually mandates the short-term approach, which subsequently spirals into more serious public health problems. For instance, if the government allowed followers of Benelles to spend insurance dollars on whole foods instead of insulin, there would be fewer dialysis clinics. Instead, the government has given the incentive to dialysis clinics over whole foods. Furthermore, the government endorsement attaches the precious placebo effect to harmful, short-term drugs, like insulin injection and uric acid reducers.
The government should reward long-term planners by recognizing the Benelles path. This would shift tax dollars from pharmaceuticals and radiation machine companies to deserving industries, like whole foods and outdoor parks, which will bring down healthcare costs and help restore some of the middle class.